Inhibition of thrombin-induced platelet aggregation by uteroglobin
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2018, Seminars in PerinatologyCitation Excerpt :A number of alternate approaches have been explored, including co-administration of piperine to enhance bioavailability, and the development of nanoparticle delivery devices.106,107 CC10, also known as uteroglobin, is a 10kDa homodimeric secretory protein produced by Clara cells in the airways and in the urogenital tract, This protein possesses a number of in vitro anti-inflammatory properties including blocking of N-Formyl-methionyl-leucyl—phenylalanine (fLMP)-stimulated neutrophil and monocyte chemotaxis,108 inhibition of phospholipase A2 (PLA2),109 inhibition of platelet aggregation,110 and suppression of IL2-stimulated production of tumor necrosis factor alpha (TNFα), IL1ß and interferon (IFN)γ in lymphocytes.111 CC10 knockout mice exposed to hyperoxia,112,113 or ozone,114 demonstrate an increased inflammatory response consisting of an exaggerated pro-inflammatory cytokine profile including IL1ß and an accumulation of inflammatory cells.
Recombinant protein 1/secretoglobin 1A1 participates in the actin polymerization of human platelets
2003, Clinica Chimica ActaAdenoviral mediated uteroglobin gene transfer to the adventitia reduces arterial intimal hyperplasia
2001, Journal of Surgical ResearchAdenoviral-mediated uteroglobin gene transfer inhibits neointimal hyperplasia after balloon injury in the rat carotid artery
2000, Journal of Vascular Surgery