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Successful treatment of progressive diffuse PEComatosis

Kay Lawson, Toby M. Maher, David M. Hansell, Andrew G. Nicholson
European Respiratory Journal 2012 40: 1578-1580; DOI: 10.1183/09031936.00076411
Kay Lawson
*Dept of Histopathology, Imperial College
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Toby M. Maher
#Interstitial Lung Disease Unit, Imperial College
¶Royal Brompton and Harefield Hospitals NHS Foundation Trust, and National Heart and Lung Institute, Imperial College
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David M. Hansell
¶Royal Brompton and Harefield Hospitals NHS Foundation Trust, and National Heart and Lung Institute, Imperial College
+Dept of Imaging, Imperial College, London, UK
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Andrew G. Nicholson
*Dept of Histopathology, Imperial College
¶Royal Brompton and Harefield Hospitals NHS Foundation Trust, and National Heart and Lung Institute, Imperial College
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  • For correspondence: a.nicholson@rbht.nhs.uk
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    Figure 1–

    Diffuse PEComatosis. a) Many of the nodules comprise cytologically bland, clear cells, while b) focally, the cells show a more spindled morphology with the nuclei being blunt-ended and containing moderate volumes of eosinophilic cytoplasm characteristic of lymphangioleiomyomatosis. Haematoxylin and eosin. Magnification: ×200 c) Computed tomography (CT) 18 months after diagnosis showed marked interlobular septal thickening in the upper lobes, a probable micronodular pattern (on this thick CT section) and a right-sided pleural effusion, but no cystic air spaces. d) 3 months later there was an improvement with less thickening of the interlobular septa (a micronodular pattern persists) and almost complete resolution of the right pleural effusion.

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Successful treatment of progressive diffuse PEComatosis
Kay Lawson, Toby M. Maher, David M. Hansell, Andrew G. Nicholson
European Respiratory Journal Dec 2012, 40 (6) 1578-1580; DOI: 10.1183/09031936.00076411

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Successful treatment of progressive diffuse PEComatosis
Kay Lawson, Toby M. Maher, David M. Hansell, Andrew G. Nicholson
European Respiratory Journal Dec 2012, 40 (6) 1578-1580; DOI: 10.1183/09031936.00076411
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