EditorialsTissue-specific glucocorticoid resistance-hypersensitivity syndromes: Multifactorial states of clinical importance☆
References (31)
- et al.
Acquired immunodeficiency syndrome-related insulin resistance and lipodystrophy: a multifactorial viral and iatrogenic condition
Endocr Pract
(2001) - et al.
p38MAP kinase-induced glucocorticoid receptor phosphorylation reduces its activity: role in steroid-insensitive asthma
J Allergy Clin Immunol
(2002) - et al.
Inhaled corticosteroids: impact on asthma morbidity and mortality
J Allergy Clin Immunol
(2001) - et al.
Alternative agents in asthma
J Allergy Clin Immunol
(2001) - et al.
Activation function 2 in the human androgen receptor ligand binding domain mediates interdomain communication with the NH(2)-terminal domain
J Biol Chem
(1999) - et al.
Glucocorticoid receptor beta: view I
Trends Endocrinol Metab
(1999) - et al.
Nuclear receptor coactivator p160 proteins enhance the HIV-1 long terminal repeat promoter by bridging promoter-bound factors and the Tat-P-TEFb complex
J Biol Chem
(2002) - et al.
Interaction of the ligand-activated glucocorticoid receptor with the 14-3-3 eta protein
J Biol Chem
(1997) - et al.
Functional modulation of the glucocorticoid receptor and suppression of NF-kappa B-dependent transcription by ursodeoxycholic acid
J Biol Chem
(2001) - et al.
Gene profiling reveals unknown enhancing and suppressive actions of glucocorticoids on immune cells
FASEB J
(2002)
Glucocorticoid and mineralocorticoid resistance/hypersensitivity syndromes
J Endocrinol
(2001)
Molecular determinants of glucocorticoid receptor function and tissue sensitivity to glucocorticoids
Endocr Rev
(1996)
Decreased corticosensitivity in quiescent Crohn's disease: an ex vivo study using whole blood cell cultures
Dig Dis Sci
(1999)
Prolonged methylprednisolone treatment suppresses systemic inflammation in patients with unresolving ARDS: evidence for inadequate endogenous glucocorticoid secretion and inflammation-induced immune cell resistance to glucocorticoids
Am J Respir Crit Care Med
(2002)
The role of stress and the hypothalamic-pituitary-adrenal axis in the pathogenesis of the metabolic syndrome: neuro-endocrine and target tissue-related causes
Int J Obes
(2000)
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