Original articlesStretch-induced morphological changes of human endothelial cells depend on the intracellular level of Ca2+ rather than of cAMP
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Transformation of keloids is determined by stress occurrence patterns on peri-keloid regions in response to body movement
2013, Medical HypothesesCitation Excerpt :Various hypotheses refer to general relationships between mechanical stresses and cellular activities. Some hypotheses explain how cells receive mechanical stresses through mechano-receptors [7–16]. Other hypotheses explain that influx of calcium ions occurs when mechano-sensitive channels are activated by mechanical stresses [17].
Corticotropin-releasing factor (CRF) can directly affect brain microvessel endothelial cells
2003, Brain ResearchCitation Excerpt :It is possible that the 2-fold induction of cAMP we detected is low because of a low affinity CRF receptor or damage to CRF receptor during the purification process. However, a 2-fold camp increase may be sufficient to initiate the signal cascade leading to morphological changes [42]. Previous studies had shown that elevations in cAMP induced the formation of tight junctions in cultured brain endothelial cells [30,41].
Studying the Mechanobiology of Aortic Endothelial Cells Under Cyclic Stretch Using a Modular 3D Printed System
2021, Frontiers in Bioengineering and BiotechnologyComparative study of variations in mechanical stress and strain of human blood vessels: mechanical reference for vascular cell mechano-biology
2020, Biomechanics and Modeling in Mechanobiology